anaerobic glycine catabolic process
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GO_0019669 |
[The anaerobic chemical reactions and pathways resulting in the breakdown of glycine, yielding energy in the form of ATP.] |
glycine catabolic process
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GO_0006546 |
[The chemical reactions and pathways resulting in the breakdown of glycine, aminoethanoic acid.] |
neurotrophin receptor activity
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GO_0005030 |
[Combining with a neurotrophin, any of a family of growth factors that prevent apoptosis in neurons and promote nerve growth, and transmitting the signal to initiate a change in cell activity.] |
signaling receptor activity
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GO_0038023 |
[Receiving a signal and transmitting it in the cell to initiate a change in cell activity. A signal is a physical entity or change in state that is used to transfer information in order to trigger a response.] |
tumor necrosis factor receptor activity
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GO_0005031 |
[Combining with tumor necrosis factor, a proinflammatory cytokine produced by monocytes and macrophages, to initiate a change in cell function.] |
death receptor activity
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GO_0005035 |
[Combining with an extracellular messenger (called a death ligand), and transmitting the signal from one side of the plasma membrane to the other to initiate apoptotic or necrotic cell death.] |
GO_0005032
|
GO_0005032 |
|
GO_0005033
|
GO_0005033 |
|
osmosensor activity
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GO_0005034 |
[Sensing extracellular osmolarity to initiate a change in cell activity, and spanning the membrane of the cell.] |
obsolete death receptor adaptor protein activity
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GO_0005037 |
[OBSOLETE. (Was not defined before being made obsolete).] |
obsolete negative regulation of complement activation, alternative pathway in another organism
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GO_0044634 |
[OBSOLETE. Any process that stops, prevents, or reduces the frequency, rate or extent of complement activation by the alternative pathway, in a different organism.] |
obsolete death receptor interacting protein activity
|
GO_0005038 |
[OBSOLETE. (Was not defined before being made obsolete).] |
obsolete positive regulation of complement activation, alternative pathway in another organism
|
GO_0044635 |
[OBSOLETE. Any process that activates or increases the frequency, rate or extent of complement activation by the alternative pathway, in a different organism.] |
obsolete death receptor-associated factor activity
|
GO_0005039 |
[OBSOLETE. (Was not defined before being made obsolete).] |
obsolete envenomation resulting in modulation of complement activation, classical pathway in another organism
|
GO_0044636 |
[OBSOLETE. A process that begins with venom being forced into an organism by the bite or sting of another organism, and ends with the regulation of complement activation via the classical pathway of the bitten/stung organism.] |
obsolete envenomation resulting in negative regulation of complement activation, classical pathway in another organism
|
GO_0044637 |
[OBSOLETE. A process that begins with venom being forced into an organism by the bite or sting of another organism, and ends with the negative regulation of complement activation via the classical pathway of the bitten/stung organism.] |
obsolete envenomation resulting in positive regulation of complement activation, classical pathway in another organism
|
GO_0044638 |
[OBSOLETE. A process that begins with venom being forced into an organism by the bite or sting of another organism, and ends with the positive regulation of complement activation via the classical pathway of the bitten/stung organism.] |
obsolete envenomation resulting in modulation of complement activation, lectin pathway in another organism
|
GO_0044639 |
[OBSOLETE. A process that begins with venom being forced into an organism by the bite or sting of another organism, and ends with the resultant modulation of complement activation via the lectin pathway in the bitten/stung organism.] |
L-methylmalonyl-CoA biosynthetic process
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GO_0019680 |
[The chemical reactions and pathways resulting in the formation of L-methylmalonyl-CoA, the L-enantiomer of 2-carboxypropanoyl-CoA.] |
L-methylmalonyl-CoA metabolic process
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GO_0046491 |
[The chemical reactions and pathways involving L-methylmalonyl-CoA, the L-enantiomer of 2-carboxypropanoyl-CoA. S-methylmalonyl-CoA is an intermediate in the beta oxidation of odd-numbered fatty acids in animals.] |